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SITO 1° CLASSIFICATO IN ITALIA 
E GIA' 1° NEL MONDO PER PROBLEMI DI MEDICINA


FREE RADICALS ARE ATOMS OR MOLECULES THAT HAVE LOST AN ELECTRON.
OXIDATION AND 'TO BE CAUSED BY MANY CAUSES:
BREATHING, METABOLISM, INFLAMMATION,
COOKING FOOD, THE LACK OF VITAMINS AND MINERALS, PESTICIDES, ASBESTOS, RADIATION RADIATION, ULTRAVIOLET RAYS VIRUSES, hydrogenated fats, heterocyclic amines, SMOKE, THE SMOG, acrylamide, PROTEIN ELICATE THE MALO dialdehyde, THE FAILURE OF ESSENTIAL FATTY THE ELECTROMAGNETIC RADIATION PRODUCED OVER THE USE OF MOBILE PHONES AND COMPUTERS. (INTER. AGENCY FOR RESEARCH OF CANCER (IARC30/7/2013), ETC.
THESE FACTORS subtract ELECTRON ATOMS AND MOLECULES TO FORM AND FREE RADICALS.
REDUCE THE FREE RADICALS EXTENDS LIFE: THE FRUIT FLIES (DROSOPHILAMEGALOBLASTER) fed DIET RICH IN ANTIOXIDANTS ARE EXPERIENCED IN OVER 66% OF 'FROM 45 DAYS TO 75 DAYS.
ATTACK THE FREE RADICALS DNA, NE oxidize COMPONENTS AND CAUSE SERIOUS INJURY, REPAIR THAT IF NOT REDUCE THE LONGEVITY 'AND MAY CAUSE CANCER.
And 'KNOWN THAT THE GAINS 2% OXYGEN FREE RADICALS PRODUCED LIZZATO AGENCIES.
ESTIMATE IS THAT FOR EVERY 10 EL. 12 OXYGEN MOLECULES IN CELLS ENTERING EACH DAY 1 OF DAMAGING THE PROTEIN 100, 1 200 ALL THE DNA DAMAGING.(ACWORTH AND COLL. ESAs, INA, 1996)
ABOUT THE 1% -5% OXYGEN USED BY MITOCHONDRIA AND 'CONVERTED INTO FREE RADICALS (ANION SUPER OXIDE (O2), hydrogen peroxide (H2O2), hydroxyl radical (OH). (Bekman KB AMES BN PHISIOL REV 78 547 - 581/1998).
The HUMAN CONSUMPTION BASED FOODS RICH IN INDUSTRIAL FATTY hydrogenated, stir-fries, PESTICIDES AND COOKED FOOD AND 'AGAINST NATURE AND' IS FULL OF FREE RADICALS AND BECOME STRONGLY OXIDANT.
A RAW FOOD TO 80%, FULL OF ANTIOXIDANTS, ELECTRON, ENZYMES, VITAMINS AND FIBER, MINERAL AND CAROTENOIDS 'NECESSARY TO COMPENSATE FOR FREE RADICALS AND REPAIR THE DAMAGE OF DNA.
IN LABORATORY RATS FED WITH RAW FOODS, RICH OF ENZYMES AND THE DURATION OF LIFE 'OF THREE YEARS, IN LABORATORY RATS fed COOKED FOOD ENZYMES AND NO' FOR TWO YEARS.
POOR DIETS OF ENZYMES shortened life 30% (DOC.HOWELL EDUARD).
ANTIOXIDANTS ARE DESTROYED BY COOKING FOR PERCENT HIGHER THAN 90%. (MORE 'ON THE PORTALS SEARCH:
(OXIDATIVESTRESS AND CANCER).
THE RIVIVENTI THEY ALL SPECIES FOLLOWING A DIET RAW 100%, FULL OF ANTIOXIDANTS AND ELECTRON, REPAIRING THE DAMAGE OF DNA.
FOR HUMANS AT LEAST TO INCREASE FOOD RAW '80% and' NEEDED TO REPAIR THE DNA AND IMPROVE THE IMMUNE SYSTEM TO VIRUSES AND BACTERIA.

 

VITAMINS, MINERALS AND REPAIR OF DNA.

THE MAN, THE RECORDS AND GUINEA PIGS DO NOT synthesize vitamin C, which is' CONTENT IN RAW FOODS.
FAILURE SUMMARY OF VITAMIN CE 'DUE TO LACK OF MAN, IN THE RECORDS IN GUINEA PIGS AND ENZYME L-GULONOLATTONE-OXIDASE.
VITAMIN CE 'WAS DISCOVERED IN 1927 BY ALBERT SZTENT GYORGY, PRE. NOBEL.
VITAMIN C (C6H8O6) E 'SOLUBLE AND' DESTROYED BY HEAT, THE KITCHEN, AND THE LIGHT FROM THE AIR, THE VITAMIN CE 'TO CONSIDER AN ELECTRON DONOR OF ASSETS.
THE VIT. C IN FACT HAVE STRONG REDUCING ACTION, CEDE ELECTRON KEEPS THE STABLE VIT. TO, AND FOLIC ACID AND B1 (thiamin).
And 'VITAL FOR MEN TAKE VITAMIN C: EACH MOLECULE OF VITAMIN C SELLS TWO ELECTRONS, dehydroascorbic acid BECOME CLEAR AND FREE RADICALS, ONCE THE VITAMIN CE' OXIDISED AND 'THE NEW REDUCED GLUTATHIONE.
THE ACTION OF VITAMIN C IN THIS VEGETABLE AND FRUIT AND 'ENHANCED BY THE PRESENCE OF flavonoids, POWERFUL NATURAL ANTIOXIDANTS.
THE VIT. C ALLOWS THE BODY USING CARBOHYDRATE, FAT AND PROTEIN AND 'LIFE FOR GROWTH AND HEALTH OF BONES, TEETH, GUMS, LIGAMENTS, ARTERIES AND COLLAGEN. (Szent Gyorgyi Albert, PR.NOBEL DIMEDICINA1937).
VITAMIN C INHIBIT THE OXIDATION OF CHOLESTEROL AND INCREASE THE PRODUCTION OF GLUTATHIONE AND CARNITINE.
THE NEEDS OF VIT. EC 'EVALUATED AT 90 MG OF, YOU CAN TO GET UP TO 2 GR. A DAY.
IN CASE OF SERIOUS CANCER THE NOBEL PRIZE PAULING PROF USED STRENGTHS OF 10 GR PER DAY WITH FAIR SUCCESS.
THE RAW MEAT CONTAINS MANY SPECIES THE VITAMINS OR PP niacin (NAD) THAT 'IS NECESSARY TO ALL OF REDOX REACTIONS. THE RAW MEAT ALSO CONTAINS MINERAL AND ENZYMES, ES. COENZYME Q10 AND THE 'POOR carcinogen.
THE NIACIN OR VIT. B3 AND 'A VITAMIN WATER-SOLUBLE, thermostable.
THE NIACIN AND HER DAUGHTER ARE nicotinamide PRECURSOR OF NAD (NICOTINE MIDEDINUCLEOTIDE) THAT MAY 'BE phosphorylated (NADP) REDUCED NADH and NADPH.
NAD TURNS ON A KEY ROLE IN THE BODIES AS A FACTOR IN PRIMARY OXIDATION REACTIONS,
JOIN IN FACT 400 ENZYMES FOR THE PRODUCTION OF ENERGY.
THE NIACIN PERFORMS A ROLE IN THE PRIMARY REPAIR OF INJURIES AND MUTATIONS precancerous DNA.
NAD AND THE 'ONE MOLECULE LIFE IN ALL THE BODIES, BOTH AS A KEY COMPONENT IN THE PRODUCTION OF ENERGY IS IN THE PROCESS OF TRANSCRIPTION AND REPAIR OF DNA. THE NIACIN AND ITS DERIVATIVES REDUCE THE RISK OF CANCER GENE P53 adjusting them SUPPRESSOR OF SKIN CANCER, BREAST, LUNG (Linus Pauling INSTITUTE OF OREGON, 2013).
NAD AND THE 'INVOLVED IN STABILITY' TELOMERE AND STABILITY IN 'THE GENOME. NAD AND THE 'APPROPRIATE FOR THE SYNTHESIS OF ENZYMES PARP (poly ADP ribose POLYMERASE). The PARP ARE 17 DIFFERENT ENZYMES WITHIN THE CELL, WHICH HAVE DIFFERENT STRUCTURE AND FUNCTION OF DIFFERENT. PARP ENZYMES ARE ESSENTIAL FOR THE REPAIR OF DNA FOR APOPTOSIS AND THE PREVENTION OF CANCER. PARP ACTIVATE ENZYMES AFTER THE DAMAGE TO DNA.
PARP ENZYMES ARE IN THE NUCLEUS, THEIR ROLE AND 'TO DISCOVER THE TEARS OF SINGLE FILAMENT (STRAND BREAKS) DNA and repair them. THE ACTIVITY OF ENZYMES AND PARP 'ALSO TIED TO LONGEVITY'. CENTENARIES ACTIVITY IN 'PARP It' VERY HIGHER THAN IN THE OTHER INDIVIDUALS, THE RISE OF LONGEVITY 'AND' DUE TO THE REPAIR OF DNA.
IN 13 SPECIES (RATS, GUINEA PIGS, RABBITS, MARMOTTE, SHEEP, PIGS, COWS, BONOBO, HORSES, ASSES, ELEPHANTS AND GORILLA MAN) THE LONGEVITY 'AND CONTENT LINKED TO DIFFERENT PARP ACTION OF DNA REPAIR. ((2008) BERSTEIN H.PAYNE AND COLL, CANCER AND AGING AS CONSEQUENCEOF unrepaired DNA).KIMURA H SUZUKI TO EDITORS
THE VIT. NAD PP O O NICOTINAMIDEADENIN DINUCLEOTIDESECONDO SCHOLARS OF THE UNIVERSITY 'OF HARVARD AND THE NEW SOUTH WALES HAS SHOWN TO HAVE A STRONG ACTION ON REJUVENATION OF MUSCLES AND ON MITOCHONDRIA OF LABORATORY MICE AND MAN. RESEARCHERS ARE ABLE TO REVERSE THE EFFECTS of aging LI CHIN IMPROVING COMMUNICATION BETWEEN MITOCHONDRIA AND THE CORE WITH A PRODUCT THAT PRODUCTION increased the NAD (SOURCE UNSW, HARVARD MED SCHOOL).
The INHIBITION OF THE SYNTHESIS OF NAD PROMOTES CELL DEATH (CURR TOP MED CHEM DOLLE ET ALL.2013, 13 (23) :2907-17)
NAD AND THE 'ESSENTIAL TO THE SYNTHESIS OF ENZYMES DEACETILANTI sirtuins. Sirtuins ARE INVOLVED IN AGING-RELATED PHENOMENA, apoptosis, AND TRANSCRIPTION OF DNA.
Sirtuins ARE SEVEN AND TAKE THE NAME TO SIR 2, A GENE THAT RULE THE METABOLISM AND DEVELOPMENT IN THE YEAST CELL. IT HAS AN INCREASE IN MAMMALS AND LONGEVITY OF SRTUINE ', ESPECIALLY OF SIR1 WITH CALORIE RESTRICTION. Sirtuins modulate MOST IMPORTANT PROCESSES AS MOBILE PHONES: DNA REPAIR, INFIAMMZIONE, CELL DEATH, AND PROLIFERATION. Recent studies suggest that sirtuins ARE INVOLVED IN THE DEVELOPMENT OF CANCER, OR ACTING AS PROMOTING SUPPRESSORS OF CANCER CELL IN ACCORDANCE WITH THE CONTEXT. (CHA YI AND ALL. BMP REP., 2013; 46 (9) :429-38.

NIACIN AND THE 'ESSENTIAL:

1) FOR THE PRODUCTION OF ENERGY
2) FOR THE SKIN PROTECTION FROM ULTRAVIOLET
3) FOR GROWTH
4) PEARL PRODUCTION OF HORMONES
5) TO REPAIR THE DNA
BEYOND THE FLESH ARE SOURCES OF NIACIN: MUSHROOMS, TUNA, THE BREWER'S YEAST, WHEAT GERM ON THE BREAST OF CHICKEN LIVER. THE EPIDEMIC OF 30 YEARS IN USA pellagra which caused 'MILLION DEAD, THE INTENSIVE USE OF BREWER'S YEAST, WHICH HAS A HIGH CONTENT OF B3 DeBello' pellagra AND EXCEPT 'MILLIONS OF LIVES. THE NEEDS OF NIACIN AND 'IN THE ADULT OF 16 MG PER DAY. THE LACK OF FOLIC ACID (VIT B 9) HUMAN CAUSES megaloblastic anemia, spina bifida AND HEART DISEASE. Folate deficiency and 'TIED TO THE DEVELOPMENT OF THE SPECIES OF COLORECTAL CANCER. Folate deficiency SADENILMETIONINA AND REDUCES DNA methylation. FOLIC ACID AND 'CRUCIAL FOR SYNTHESIS AND DNA REPAIR. THE LACK OF FOLIC ACID ACCUMULATION OF ACTS AND BREAKING OF CHROMOSOMES uracil. (SUSAN J DUTHIE UN.ABERDEEN).
THE VITB12 PERFORMS A KEY ROLE IN THE SYNTHESIS OF DNA IN GROWTH AND DEVELOPMENT IN METABOLISM AND AMINO ACIDS IN THE HEALTH OF NERVES, RED BLOOD CELLS, SKIN. THE NEEDS OF B12 AND '2.4 mcg PER DAY. MORE SOURCES 'B12 ARE IMPORTANT: LIVER, MEAT, FISH, EGGS MILK. The VEGETARIAN ARE PREPARED TO LACK OF B12. B12 DEFICIENCY LEADS pernicious anemia and neuritis. VITAMIN B2 (Riboflavin) 'WAS ISOLATED IN 1927 BY PROF. PAUL GYORGY.TRASPORTATATI BLOOD TO LIVER IS TRANSFORMED INTO TWO COENZYMES ESSENCE OF REDOX REACTIONS IN ENERGY METABOLISM: FMN (FLAVINMONONUCLEOTIDE) and FAD (FLAVIN adenine DINUCLETIDE).
THE VIT B2 IS IN VEGETABLES IN YEAST IN MILK IN 'EGG.
DEMAND AND '0.6 mg/1000 CALORIE PRO DIE. Riboflavin AND THE 'ESSENTIAL:
1) ENERGY PRODUCTION
2) FOR GROWTH
3) TO IMPROVE THE IMMUNE SYSTEM.
4) FUNCTION FOR THE HORMONE.
Heterocyclic amines
MEAT COOKED TO HIGH TEMPERATURE AND LONG OXIDIZES, heterocyclic amines DEVELOP AND BECOME CARCINOGENIC glycated proteins, SO PROTEIN AND DNA DAMAGE THE BODY AND CAUSE MUTATIONS AND CANCER. (SEE HETEROCYCLIC AMINES AND DNA DAMAGE.) Jpn J CANCER RES MARS 1999, 90 (3) :268-275).
Heterocyclic amines (AM 17) COOKING WITH LONG FORM OF MEAT, POULTRY AND FISH HAVE CANCER AND FORM FOR REACTION BETWEEN AMINO ACID CREATINE AND THAT IS COOKING WITH PROLONGED AND HIGH TEMPERATURE. (MAIL FROM THE NATIONAL CANCER CANCER INST. U.S.).
The EATING MEAT COOKED IN THOUSANDS OF STUDIES CONDUCTED IN THE WORLD, HAVE INCREASED RISK OF STOMACH CANCER OF THE COLON, KIDNEY, WITH RESPECT TO THE BREAST AND PANCREATIC VEGETARIAN. (Nat. CANCER INST-USA). PROTEIN OF EGGS, MILK, TOFU, VEGETABLES AND LIVER NOT CONTAIN CREATINE AND OR NOT GROW GROW VERY FEW heterocyclic amines. (HETEROCYCLIC AMINES ANDCANCER. Wilkipedia)
A CONSUMING 5000. ORAC (Antioxidants) PER DAY SO DRAMATIC REDUCES THE IMPACT OF CANCER, HEART DISEASE,
M.DIALZHEIMER, M. PARKINSON'S INFECTIONS ETC, ACCORDING TO the Depart. AGRICULTURE USA.
And 'THAT WAS CALCULATED 40 NUTRIENTS ARE REQUIRED TO HUMAN CONSUMPTION. The PIU 'IMPORTANT ARE 8: VIT B12, FOLIC ACID, VIT.B6, PP VIT, VIT C, VIT E, ZINC, IRON. MIMA THEIR LACK OF RADIATION EFFECTS ON DNA. Folate deficiency, OF B6, B12 LEADS TO CHROMOSOME BREAKAGE TO BUILD IN uracil DNA. EPIDEMIOLOGICAL STUDY REPRESENTS AN INVERSE RELATIONSHIP BETWEEN THE INTRODUCTION OF FOLIC ACID AND CANCER OF THE COLON. Rats fed diets low in folate, methionine and choline FOR 10 WEEKS HAVE BEEN MARKED THE DNA DAMAGE OF CELLS OF THE COLON. (PMID: 10918992) THE LACK OF FRUITS AND VEGETABLES CAUSES DAMAGE EQUAL TO RADIATION. (HOSPITAL OAKLAND RESEARCH INSTITUTE 2005). STUDIES HAVE SHOWN THAT THE SYNTHESIS OF DNA AND 'SLOWED TO A LOW LEVEL OF MAGNESIUM.
MAGNESIUM STABILISES THE DNA POLYMERASE AND COENZYME OF THE DNA REPAIR BY MUTAGENIC. MAGNESIUM IN CONJUNCTION WITH atypical PARTICIPATE IN THE SYNTHESIS OF RNA. THE LACK OF VITAMIN D3 ACCORDING TO SOME RECENT STUDIES CAN INCREASE THE RISK OF CANCER UP TO 77%. (AMERICAN JOURNAL OF CLINICAL NUTRITION), ANTI CANCER RESEARCH).
THE VIT. D3 E 'MIRACULOUS IN THE PREVENTION OF BREAST CANCER PREVENTION WITH UP TO 50% OF CASES AND PREVENTION WITH UP TO 2/3 OF CASES OF CANCER COLORECTAL. FOR MORE 'SEARCH ON THE WORLD PORTALS (CANCER AND VITAMIN D). THE HUMAN DNA SUFFERS INJURY ON AVERAGE 19,200 PER DAY, THAT COME UP TO 1,000,000 A DAY OF INJURY IN THE EVENT OF OXIDATIVE STRESS: THE REPAIR OF DNA DAMAGE AND 'ESSENTIAL HEALTH AND THE PREVENTION OF DISEASE AND CANCER.
THE DNA IN ADDITION TO TRANSMIT CHARACTER HEREDITARY, plays a decisive role TO RENEW THE LIVING CELLS BY INSERTING THE AMINO CONTINUOUSLY AND BE DIFFERENT FOR EACH.
THE DURATION OF LIFE CELL AND 'OTHER:

  • WHITE BLOOD CELLS: 10 HOURS / DAYS 3
  • RED BLOOD CELLS 4 WEEKS
  • PLATES: 10 DAYS
  • SPERM: 2/3 DAYS
  • COLON CELLS: 3/4 DAYS
  • Epidermal cells: 2/4 WEEKS.
  • BONE CELLS: 25/30 YEARS
  • LYMPHOCYTES: 2 MONTH / YEAR 1.

INJURIES OF RISING DNA aging, FOR EXAMPLE IN LYMPHOCYTES OF THE PERSONS 70 YEARS OF THE PERCENTAGE OF DNA DAMAGE AND 'BY 50% IN PATIENTS OF 60/69 YEARS AND' THE 41%. (PMID: 11337012). INJURIES OF DNA FROM ENVIRONMENTAL OR OTHER CAUSES BEYOND REPAIR GIVE RISE TO MUTATIONS THAT CAUSE CANCER AND LEUKEMIA. A RECENT RESEARCH IN THE U.S. HAS IDENTIFIED 74 REGIONS OF DNA ASSOCIATED WITH PROSTATE CANCERS OF THE BREAST OF THE OVARIES. IN THIS RESEARCH AND 'WAS STUDIED THE GENOME OF SUBJECTS 250000.
CELL DAMAGE AND ESSENTIAL FATTY ACIDS
CAUSES OF CELLULAR AND MITOCHONDRIAL DAMAGE ARE: LACK OF OXYGEN, POWER IMBALANCE, TOXIC FOOD ALLERGIES, RADIATION, INFECTIONS, DEFICIENCY DiGrassi POLYUNSATURATED THOSE ESSENTIAL ETC. THE FAILURE OF ESSENTIAL FATTY ACIDS OMEGA-3 AND OMEGA-6, THE MAN WHO CAN NOT SYNTHESIZING DIRECTLY AFFECT THE CELL MEMBRANE, MEMBRANE LIPIDS IN FACT ARE MADE OF THESE. IF THE ESSENTIAL FATTY ARE ABSENT FROM POWER CELLS SHOULD REPLACE THESE FATS WITH SATURATED FAT, YOU ALTER THE MEMBRANES, BECOME LESS PERMEABLE, RECEPTORS AND THEY WILL REDUCE THE BEGINNING OF MANY DISEASES ES. 2 DIABETES, THE DNA DAMAGE AND CANCER. THE FAILURE OF ESSENTIAL FATTY LOCK MANY FEATURES REDUCE THE PASSAGE OF VITAL NUTRIENTS AND OXYGEN, DETERMINING, ISCHEMIA AND ASFISSI acellular.
The fatty acids OMEGA 3 AND OMEGA 6 ARE REQUIRED FOR THE SYNTHESIS OF PROSTAGLANDINS. PROSTAGLANDINS ARE THE STANDARD 1,2, 3. THE OMEGA 3 PROMOTE THE SYNTHESIS OF PROSTAGLANDINS SERIES 1 AND 3 anti-inflammatory and anti-platelet vasodilator, hypotensive PROMOTE THE OMEGA 6 SUMMARY OF SERIES 2 PROSTAGLANDINS constrictor binding and PRO AND INFLAMMATORY.
ANTI CANCER FOODS.
CURRENTLY IS CALCULATED THAT 35% OF CASES OF CANCER IS CAUSED BY CAUSE ALIMENTARI.L 'SUMMARIZING MAN NOT THE ESSENTIAL FATTY ACID, VITAMIN C, folate, LA VIT.B12 THE VIT A, VIT E. THE L' RECRUITMENT OF THESE NUTRIENTS AND 'ESSENTIAL. , Cancerous cells proliferate ACID IN ENVIRONMENT AND ENVIRONMENT FULL OF SUGAR, THEREFORE, IT IS RECOMMENDED THAT A POWER ALKALINE AND POOR OF SUGAR. FOLIC ACID CARRIES A KEY ROLE IN DNA REPAIR AND PREVENTION OF GASTRIC CANCER AND COLON.
INCREASE IN FACT, DNA methylation, REDUCES THE INCREASE IN uracil DNA PREVENTS THE DESTRUCTION OF THE CHROMOSOMES AND INFLAMMATION HELICOBACTER. The FOLATE ARE DESTROYED BY 50 TO 90% OF THE COOK AND THE 70% OF THE LIGHT. FOODS RICH IN NUCLEOTIDES ARE PREFERRED FOR THE SYNTHESIS AND REPAIR OF DNA. The NUCLEOTIDES ARE THE SUB UNIT 'WITH WHICH THE BODY AND CREATE DNA RNA. MORE FOOD 'RICH ARE NUCLEOTIDES: BREAST MILK, BREWERS YEAST, sardines, lentils, beans, OYSTERS, AND SPIRULINA CHLORELLA. AMONG THE MOST FOOD 'HYDROGEN RICH'S' THE WATER, TWO LITRES PER DAY ARE ESSENTIAL. RICH FOODS OF ELECTRONS OF OMEGA 3 FATTY VITAMINS ARE: The FLAX SEED OIL, CITRUS, sprouted seeds, BEET THE SPIRULINA, GARLIC, ALMONDS, NUTS, SEEDS, CARROTS, ETC .
MORE 'FOODS CONTAINING VITAMINS AND MORE ELECTRONS' INCREASE THE PRODUCTION OF ENERGY. THE BODY IN FACT USED TO PRODUCE THE ELECTRON ENERGY: ELECTRONS ARE PROCESSED THROUGH THE SYSTEM IN atypia CYTOCHROME / OXIDASE IN CELL MITOCHONDRIA.
FOR THE PREVENTION AND TREATMENT OF CANCER ARE PREFERABLE FOODS RICH IN VITAMIN C, E, A, B6, B12, D3, and folate, OMEGA 3 FOUND IN RAW FOODS: LEMON, GRAPEFRUIT, ORANGE, STRAWBERRIES, SPINACH, CITRUS, ASPARAGUS , CRUDE VEGETABLE LEAF GREEN, CELERY, ALGAE (CHLORELLA), UVA, FIGS, CUCUMBER, MELOGRANI AND RASPBERRY (FOR ACID CONTENT IN ELLAGIC) CURRY, WHEAT GERM, BREWERS YEAST, FLAX SEEDS, Pomidori rich in lycopene, ETC . And 'PROFIT MAKING BUSINESS' TO INCREASE OXYGEN AVAILABLE PHYSICS.
THE FRUITS OF THE MOST 'ALKALINE ARE: FIGS, APPLES, GRAPES, BANANAS, MELONS, MANDORLE.E' SHOULD REDUCE THE COOKED FOOD:, cancerous cells proliferate in an acidic environment, OXYGEN POOR, THE COOKING ACIDOSIS CAUSES AND LOSS OF OXYGEN. (PROF. O.WARBURG, PR.NOBEL MEDICINE 1931). REDUCE FOOD RICH IN SATURATED FAT AND FRIED: FAT MILK CHEESE AND MEAT COOKED PASTEURIZED, BOTH GROW AND lipoperoxides malondialdehyde. MORE FOOD 'ACIDS ARE: CHEESE, WHITE BREAD, SUGAR, COCA COLA, YOGURT AND JUICES SUGAR, MEAT, FISH, CHICKEN, DESSERT. And 'SUGGESTED TAKING OF WATER (2 LITRES PER DAY), RAW FRUIT (5 PORTIONS OF AL), THE RAW VEGETABLES (5 PORTIONS OF AL), FLAX SEED OIL RICH IN OMEGA-3, DRIED FRUITS RAW (RAISINS , APRICOT, DRIED FIGS), SOYA BEANS, BEANS AND PEAS SPECIES or sprouted, PEAS, EGGS (WEEK 2), TOFU, YOGURT LEAN, BOWS OF MILK 2% MILE, THE WHOLE TOOTH PASTE AND RICE IN SMALL QUANTITIES , POTATOES, RAW FISH OR seared FOR A FEW MINUTES, ALMONDS, DATES, CHESTNUT, EXTRA VIRGIN OLIVE OIL IN SMALL QUANTITIES 'BETWEEN THE PREFERRED VEGETABLES BROCCOLI, CABBAGE, CAULIFLOWER. (RICH AND INDOLE sulforaphane). USE SALT IN MINIMUM QUANTITY, AVOID FOODS RICH IN SALE, USE GARLIC, ONIONS AND SPICES IN LARGE QUANTITY '. BETWEEN THE SPICES ARE USEFUL AND CAPERS ORIGANO THAT ACCORDING TO BLOCK THE RECENT SEARCHES MALO dialdehyde.
AMONG THE MOST FOOD 'HARMFUL will be:

  • GENETICALLY MODIFIED FOODS.
  • BEEF AND PRESERVED, WHETHER containments nitrites and nitrates.
  • THE REFINED FLOUR, impoverished OF VITAMINS, MINERALS AND FIBRE CASE THOSE HIGH GLYCEMIC INDEX. SUGAR FEEDING OF CANCER CELLS DIRECTLY AND THEY PROMOTE THE METASTASES.
  • SUGAR REFINED FOODS CONTAINING SUGAR REFINED.
  • APPLES, GRAPES STRAWBERRIES HARMFUL IF TREATED WITH PESTICIDES.
  • SALMON FARMING IF TREATED WITH carcinogenic PCBs, PESTICIDES AND ANTIBIOTICS.
  • FATS hydrogenated, CAUSING SERIOUS INJURY TO THE CELL MEMBRANE.
  • SWEETENERS OF VARIOUS (Aspartame, Saccharin,) ACCORDING TO RESEARCH THE 'EFSA (Eur FOOD SAFETY, AUITORITY) MAY INFLUENZARELO DEVELOPMENT OF CANCER. SOURCE 1) CARE COM 2. 2) www.natural NEWS.COM / 021 808-CANCER PREVENTION).

AVOID high-calorie foods, REDUCE THE FATTY FOODS TO 20% OF THE TOTAL CALORIES, CALORIES JOGGING WITH THOSE UNDERTAKEN IN OUTPUT.
DNA DAMAGE, AND CANCER acrylamide
Acrylamide is' A COMPOUND WITH CHEMICAL FORMULA: C3H5NO, It is known neurotoxic AS A LETHAL AND A STRONG carcinogenic in laboratory animals. IN 2002 ITS DISCOVERY IN SOME COMMON FOOD IN THE OVEN BAKED OR FRIED (BREAD, CRAKERS, TOAST, CAKES, COOKIES, POTATOES) SCIENTISTS FROM SWEDEN HAS HAD A HUGE MR. L 'acrylamide NOT' BEEN FOUND IN FOODS OR BOILED IN FOODS RAW. Acrylamide is formed FROM THE REACTION BETWEEN 'asparagine AMINO ACID AND GLUCOSE FRUCTOSE WITH COOKING AT HIGH TEMPERATURES ABOVE 120 DEGREES FOR A LONG TIME. Acrylamide IS ALSO IN THE CAFFE 'IN THE PRUNES IN ALMOND ROASTED IN BLACK OLIVES IN WHOLE BREAD. CIGARETTE SMOKING AND 'A BIG SOURCE OF acrylamide EE' BEEN SHOWN THAT INCREASES THE LEVEL IN THE BLOOD acrylamide THREE TIMES MORE FOOD COOKED. (WIKIPEDIA) 's acrylamide exposure CAUSES DAMAGE TO THE GERM CELLS OF MALE RATS, INDUCING REDUCED FERTILITY' INCREASED MORTALITY AND 'THE SAME RESULTS AND THE SAME DOSES ARE occurring in humans. (NIXON BJ AND COLL. UNIV OF NEWCASTLE, AUSTRALIA CALLAGHAN, 2012 SEP, 129 (1) :135-145).
THE HUMAN LYMPHOCYTES IN A DERIVATIVE GLICIDAMIDE acrylamide CAUSED SERIOUS DAMAGE TO DNA binding to adenosine and explicit GUANINE It act as powerful carcinogens. AND JUST AS 'OCCURRED IN THE CELLS WITH TESTIS NET REDUCTION OF FERTILITY'. (HELLAND HANSEN, Norvegian RESEARCH COUNCIL IN EUROPEAN PROJECT: HEATOX -2011) (FOR MORE 'SEARCH ON THE WORLD PORTALS: BREAD AND CANCER, 43.5 million ARTICLES ON GOOGLE COM, acrylamide AND CANCER, ETC.)

Malondialdehyde (MDA), DNA AND CANCER

Malondialdehyde AND 'THE BEST' IMPORTANT PRODUCT OXIDATION OF FATTY POLYUNSATURATED MAY 'HAVE ALSO BY ORIGIN AND PROSTAGLANDINS deoxyribose. MDA HAS THE FORMULA 2 (CHO) 2 AND 'A REACTIVE ALDEHYDE. THE CASE MDA CYTOTOXICITY 'oxidizing MEMBRANE FATTY AND REACTS WITH deoxyadenosine AND CREATING DEOSSIGUANINA DNA mutagenic compounds and carcinogens. MDA IS LOCATED IN THE PLASMA CONCENTRATION OF 1 mM AND CONCENTRATION IN URINE IN 0-3 mM. MDA AND THE 'undetectable.
MDA AND THE 'REGARDED AS A MARKER OF OXIDATIVE STRESS. THE COOKING OF PALM OIL AND SUNFLOWER SEEDS GROW MDA. ORIGANO AND CAPERS IN SOME RESEARCH STUDIES HAVE SHOWN TO OPPOSE THE DAMAGE CAUSED MDA. UNOSTUDIODELL'UNIV. MUKKA OF (INDIA) CHECKED THE RELATIONSHIP BETWEEN THE LEVEL OF MDA EVIT. AECI N80 HEALTHY SUBJECTS WITH OR oropharyngeal cancer. MDA WAS THE MOST 'HIGH LEVEL OF VITAMINS AND MORE' LOW IN SUFFERING FROM CANCER. (ASIAN PAC. CANCERPREV.2012 J., 13 (11) :5763-5) The ASSUMPTIONS PROF. JAMES WATSON SUGLIANTIOSSIDANTI AND CANCER.
RECENTLY THE NOBEL PRIZE 1962PROF. JAMES WATSON HAS MADE AN ANALYSIS NOTE TO THE EFFECT THAT THE SYNTHETIC ANTIOXIDANTS AND ABOVE ALL THOSE COULD REDUCE THE EFFECTIVENESS OF CHEMOTHERAPY AND RADIOTHERAPY IN THE TREATMENT OF CANCER.
THIS REFERS TO THE ANALYSIS OF CANCER THERAPY AND DOES NOT AFFECT THE PREVENTION OF CANCER. CONTRARY TO THE ABOVE ANALYSIS WITH MANY OTHER CONSIDERATIONS AND EXPERIENCES OF OPPOSITE NATURE.

FREE RADICALS

The PIU 'COMMON OR FREE RADICALS (ROS REACTIVE OXYGENSPECIES), ATOMS OR MOLECULES THAT HAVE LOST ONE OR MORE OF' POSITIVE ELECTRON AND OWN ELECTRIC CHARGE ARE:

  • L '= O2 ANIONESUPEROSSIDO
  • Hydroxyl radical OH =
  • Hydrogen peroxide = H2O2 (hydrogen peroxide)
  • THE PEROXONITRITE = ONOO

The PIU 'PERICOLOSIPER ARE THE DNA: RADICAL hydroxyl (OH) AND PEROXONITRITE (ONOO), PRODUCED BY THE REACTION = NO + O2 = ONO2. DAMAGE INDUCED DALL'OSSIDRILE OH ARE MORE 'SERIOUS: OH FACT HAS A VERY SHORT SHELF LIFE, HAS A HIGH REACTIVITY' AND 'OXIDIZES DANGEROUS BECAUSE THE BASIS OF 4 DNA, deoxyribose, PROTEIN AND FAT.

THE OH RADICAL IS ACTING IN FOUR WAYS:

  • REMOVAL OF HYDROGEN: OH + CH3OH + H2O = CH2OH.
  • ADDITION OF HYDROGEN: = OH + C6H6 (OH) C6H6
  • ELECTRON TRANSFER: OH + (FE (CN) 6) 4 = FE (CN) 63 + OH.
  • INTERACTION OF RADICALS: OH + OH = H2 O2

OH AND FOR THE PRODUCTION OF 'THE NECESSARY PRESENCE OF IRON BY THE REACTION OF FENTON: FE (2) + H2O2 = FE (3) + OH + OH. THE WORKS oxidizing PEROXINITRITE A BASE OF THE DNA GUANINE. 'WAS CALCULATED THAT THE OH RADICAL MOLECULES ON ACTING WITHIN A DIAMETER OF 3 AND PEROXINITRITE NMS NMS IN 9.
THE PRODUCTION OF FREE RADICAL SUPER OXIDE O2 IS CONSTANTLY IN AEROBIC METABOLISM.
UNDER NORMAL CONDITIONS AND EVERY BODY CELL 'EXPOSED A10.EL.10 SUPER OXIDE MOLECULES EVERY DAY FOR AN INDIVIDUAL TO 75 KG CALCULATE AN EXHIBITION OF 2 KG OF SUPER OXIDE YEAR. (PROF BALZ FREI DIR.CENTRO Linus Pauling. U.S.) The INSULT TO OXIDATIVE DNA AND 'LINKED TO CANCER, THE INSULT TO OXIDATIVE LDL CHOLESTEROL AND' arteriosclerosis TIED THE INSULT TO MITOCHONDRIA AND OXIDATIVE 'TIED TO THE REDUCTION OF ENERGY PRODUCTION, THE INSULT TO OXIDATIVE AND TELOMERES 'TIED TO LONGEVITY'.

HYDROGEN AND OXYGEN

HYDROGEN MORE 'TO OXYGEN AND' THE REAL FUEL OF LIFE (Albert Szent-GYORGY P. NOBEL).
HYDROGEN AND OXYGEN IN PARTICIPATING IN A CONTINUOUS CYCLE THAT CREATES THE LIVING BODIES OF WATER AND ENERGY.
IN OXIDATION REACTIONS OF HYDROGEN AND 'AN ESSENTIAL OF ELECTRON DONOR AND OXYGEN AND' THE ESSENTIAL electron acceptor.
THE MORE 'A BODY OR A FOOD AND' RICH IN ELECTRON AND MUCH MORE HYDROGEN 'RESISTANCE TO OXYGEN FREE RADICALS AND LONG LASTING. HYDROGEN FROM WATER COMING, FROM THE AIR FROM CARBOHYDRATES, PROTEINS AND FATS FROM AND 'DELIVERED IN MITOCHONDRIA WITH A DEFINED PROCESS HYDROGEN SHUTTLE. HYDROGEN AND 'THE CELL USED TO PRODUCE atypia, THE FUEL OF LIVING ORGANISMS.
DEPOSIT IS PART OF HYDROGEN IN QUANTITY 'DIFFERENT (HYDROGEN POOLING) IN VARIOUS BODIES (SZENT-GYORGY PR. NOBEL). 1THE LIVER CONTAINS MORE 'BIG POOL OF HYDROGEN, THE LOWER THE SPLEEN. STRESS, AND THE JUNK FOOD pollution DECREASE THE POOL OF HYDROGEN IN 'BODY AND MANY PERSONS, ESPECIALLY THE ELDERLY ARE HYDROGEN depleted. (SZENT-GYORGY ALBERT PR. NOBEL)
OXYGEN BURNING IN CELLS TO PRODUCE HYDROGEN ENERGY REQUIRED TO LIFE. RESEARCH HAS SHOWN THAT MANY BODIES IN CELLS AND HYDROGEN RETAIN THE DEPLETION OF HYDROGEN OR IMPLY THE AGING AGING.
DNA IN HYDROGEN HYDROGEN BONDS THROUGH THE CONNECT THE MOLECULES OF THE DOUBLE HELIX. AGING WITH THESE LINKS AND MODIFYING THE LACK OF HYDROGEN DAMAGE AGGRAVATES.
FOOD, WATER AND AIR ARE GREAT SOURCES OF HYDROGEN, ESPECIALLY THE CARBS ARE MORE 'BIG DONORS OF HYDROGEN (SZENT-GYORGY ALBERT PR NOBEL)
L 'HYDROGEN HAS A KEY ROLE IN THE PRODUCTION DELL'ATIPI THE FUEL OF' BODIES. HYDROGEN FOOD PROVIDES THE NECESSARY IN PROTON AND ELECTRON TRANSPORT CHAIN ??MITOCHONDRIAL ELECTRON. COOKING DELETE MOST OF ELECTRON AND HYDROGEN BY THE FOOD AND BODIES OF HYDROGEN depleted. HYDROGEN CARRIES A STRONG ACTION
ANTIOXIDANT IN TISSUE.
WORK IN A JOURNAL PUBLISHED BY CLIN. BIOCHEM NUTR.DEL March 2010 46 (2) ATAUNORI NAKAO THE AUTHORS AND COLL. Administering HYDROGEN RICH WATER FOR 8 WEEKS HAVE CAUSED AN INCREASE IN THE SOD (superoxide dismutase) OF 39% AND A REDUCTION OF TBARS (DERIVATIVES OF lipoperoxides) of 43% in the urine.
HUNZA LIVES OF THE PEOPLE TO THE SLOPES OF THE HIMALAYAS AND 'NOTE FOR THE GREAT NUMBER OF CENTENARIES AND FOR A DIET RICH EXCLUSIVE NATURAL FOODS HAS NO JUNK FOOD.
GLIHUNZA GLACIER WATER USE, AND THAT 'STRONGLY ALKALINE VERY RICH IN ACTIVE HYDROGEN (HYDROGEN WITH AN ELECTRON IN PIU'), HIGH IN MINERALS AND HAS STRONG ANTIOXIDANT ACTION.
WATER USED BY HUNZA HAS THESE FEATURES
1) HAS A HIGH ANTIOXIDANT POWER: ORP = 800.
2) HAS A POWER 8 TIMES THE ANTIOXIDANT VITAMIN C IN COENZYME Q10, Pycnogenol AL.
3) alkalizes FABRICS.
4) PROMOTES HYDRATION AND INTRA extracellular.
5) ON PROTECTION AGAINST OXIDATIVE STRESS.
6) REPAIRS AND PROTECTS THE TELOMERES.
7) IMPROVES MITOCHONDRIAL MEMBRANE POTENTIAL.

DEFENSES OF BODIES BY FREE RADICALS

TO RESIST THE OXIDATIVE DAMAGE CAUSED DALL'ANIONE SUPEROXIDE ROS AND THE OTHER HAS THE NATURE WITH LIVING ORGANISMS (PLANTS AND ANIMALS) OF AN ASSEMBLY OF ANTIOXIDANT ENZYMES, ELECTRON DONORS AND AS DEFENDING THE OXIDATION DNA, PROTEIN, FATS CARBOHYDRATES AND CELL. BY THE TIME THE RATE OF ANTIOXIDANT ENZYMES IS ALWAYS MORE CUTS ', IS REDUCED THE DEFENSES OF CELLS IN LIEU OF ELDERLY AND OXIDATIVE DAMAGE.

ANTIOXIDANT ENZYMES ARE:

1) O superoxide dismutase SOD.
2) CATALASE.
3) GLUTATIONO PEROXIDASE.

THE SOD (superoxide dismutase)

SOD THE CONTRARY THE FREE RADICAL SUPEROXIDE O2, UDG IN OE H2O2 (hydrogen peroxide) (SECOND LAREAZIONE:
O2 O2 +2 H + = O2 + H2O2. THE HYDROGEN PEROXIDE (H2O2), superoxide anion E'MENO REACTIVE BUT 'IN THE PRESENCE OF ORIGIN IRON IN the hydroxyl OH. SOD WAS THE DISCOVERY BY PROF. Irwin Fridovich OF DUKE UNIVERSITY IN 1968.THE SOD PROTECTS THE DNA AND PROTEIN OXIDATION. SOD AND ACTS AS THE ANTIOXIDANT COMEANTIINFIAMMATORIO.
WEAKNESSES OF THE SOD may contribute to 'ALS (amyotrophic lateral sclerosis) Or Lou Gehrig's disease. THE SOD IS IN GOOD AMOUNT 'IN THE MELONS, BROCCOLI, BRUSSELS SPROUTS IN, IN SOY SPROUTS, CABBAGE IN, IN CARDI AND IN MANY PLANTS, EVEN IN THIS EVENT TOGETHER TO DEFEND VIT CI FRUIT, LEAVES, FLOWERS, PLANTS DALL'ANIONE SUPEROXIDE.

HUMANS ARE 3 TYPES OF SOD:

1) THE MITOCHONDRIAL
2) THE CYTOPLASMIC
3) extracellular.

GENES ARE LOCATED IN CHROMOSOMES OF SOD AND 21.6 4.
SOD CONTAINS THE MITOCHONDRIAL MANGANESE as a cofactor, THE OTHER TWO COPPER AND ZINC CONTAINING as a cofactor.
MISSING THE RATS OF SOD (2) MITOCHONDRIAL DIE A FEW DAYS AFTER BIRTH TO THE STRONG OXIDATIVE STRESS.
SECOND STUDY OF THE NATIONAL INSTITUTE OF HEALTH USA LEVELS ARE RELATED TO LONGEVITY SOD 'OF DIFFERENT SPECIES AND INDIVIDUALS. THE MAN HAS THE LEVEL MORE 'HIGH SOD OF MAMMALS. (90MICROGRAMMI FOR ML).
THE CHIMP 'HAS LEVELS FOR 40 THOUSAND LIVES MICROGRAMS META' ABOUT MEN. IN THE FRUIT FLY (DROSOPHILA) E'RIUSCITI IS A DOUBLE THE CONTENT OF GENETICALLY SOD CELL, AND ALSO OF 'THE INCREASED LONGEVITY', DROSOPHILA THE POOR OF THE BIRTH OF SOD SOD HAS A DRAMATIC life shortening.
IN PLANTS AND THE SOD 'widespread IT WORKS BY ANTIOXIDANT PROTECTING PLANTS FROM ROS (RADICALS)
SOD AND THE 'LIKE A VERY POWERFUL ANTIOXIDANT ENZYME IS ESTIMATED 3500 TIMES MORE' THE VITAMIN C LEVELS OF GENETICALLY SOD UP TO 50% MAY VARY FROM ONE TO ANOTHER MAN AND THIS EXPLAINS THE LONGEVITY 'DIFFERENT AND THE DIFFERENT RESISTANCE DISEASES.
THE SOD 'WAS USED TO treat arthritis, CORNEAL ULCERS OF MALATTIEINFIAMMATORIE, RADIATION TO HEAL WOUNDS AND BURNS.
SOD FOR THE MOUTH AND 'DISTRUTTADALL'AC. Hydrochloric
STOMACH.
A SUPPLEMENT THAT 'MELONS AND CONSISTS OF AN EXTRACT OF WHEAT, GliSODin THE INCREASE OF 90% OF THE PRODUCTION SOD, BECAUSE IT INCREASES ABSORPTION OF MELON 10 TO 80 MINUTES.
CATALASE. CATALASE AND THE 'A POWERFUL ANTIOXIDANT ENZYME, E'FORMATA FROM 4 polypeptide chains, EACH CONTAINING AMINO 500; CATALASE It has been THE DISCOVERY IN 1811 BY LOUIS Thénard.
CATALASE CONTAINS THE 4 GROUPS porphyrin EME ALLOWING THE CATALASE TO REACT WITH H2O2 (hydrogen peroxide).
Catalase breaks down hydrogen peroxide H2O2 AND OXYGEN IN WATER, THIS PROCESS IS IN SUCH PERISSOSOMI CYTOPLASM corpuscles.
EACH MOLECULE CATALASE CONVERTE 40 MILLION OF MOLECULAR HYDROGEN PEROXIDE IN WATER PER SECOND, LIKE THIS PROTECTS CELLS PEROXIDE FROM WATER, THE BODY PRODUCES NO STOPS.
THE CYANIDE AND 'A POWERFUL INHIBITOR CATALASE AND THEY LOCK ACTION.
CATALASE AND THE 'widespread PLANT AND ANIMAL IN THE WORLD, MUSHROOMS ARE PRODUCERS, ITS EASY TO DEFICIENCY The ONSET DIABETES IN MAN 2. ACCORDING TO RECENT RESEARCH OF LOW-LEVEL CATALASE AFFECTING HUMANS 'graying of hair AND AGING.
STUDIES UNIVERSITY 'OF SEATTLE WASHINGTONIN INDUCED IN RATS AN INCREASE IN THE CATALASE.
WITH THE INCREASE OF LONGEVITY CATALASE 'IN MICE E'AUMENTATA 20%, ABOUT 25 YEARS IN MORE' IN HUMANS.
PLANT IN THE WORLD GREEN LEAVES are rich in SOD and catalase, WHEN THEIR RATE IS LOWER THE LEAVES ENZYME turn yellow and die. A NEW PRODUCT THE GliSODin, MELONS AND AN EXTRACT OF WHEAT SLOW ABSORPTION OF CATALASE MELONS OF 10 TO 80 MINUTES AND LEADS TO AN INCREASE IN THE CATALASE 171%.

 

GLUTATIONOPEROSSIDASI

THE SYSTEM OF GLUTATHIONE AND 'ESSENTIAL FOR LIFE, A LACK OF GLUTATIONOSINTETASI LEADS TO PREMATURE DEATH FOR BRAIN OXIDATIVE DAMAGE. THE ADMINISTRATION OF GLUTATHIONE IN RATS WITH LIVER CANCER HAS CAUSED THE REGRESSION OF TUMORS 81% of cases. (NOVI AMSCIENCE 2012/1981 :541-2)

GLUTATIONOPEROSSIDASI PERFORMS THE TWO VITAL FUNCTIONS:

  • H2O2 TRANSFORMS INTO WATER IN CELLS
  • I FOUGHT lipoperoxides ELIMINATING A WIDE RANGE OF FREE RADICALS.

THE REACTION CATALYZED BY GLUTATIONOPEROSSIDASE AND ':
2GSH + H2O2 = GS-GS +2 H2O
GLUTATIONOPEROSSIDASI DOES THE ELECTRON TRANSFERRING FROM THE GLUTATHIONE lipoperoxides EA neutralizing H2O2.
ARE THERE IN 'MAN OF 8 DIFFERENT ISOFORMS GLUTATIONOPEROXIDASE
The ENZYME AND 'MADE UP OF THREE TO FOUR AND AMINO ACID MOLECULES ATOMS OF SELENIUM. THE SELENIUM AND 'AN ESSENTIAL FOR GLUTATIONOPEROSSIDASI, IN REGIONS WHERE THE QUANTITY TERRENOCONTIENEGRANDI' OF SELENIUM, EXAMPLE THE SOUTH DAKOTA, CANCER AND 'VERY RARE.
THIS SHOWS THAT DEFENDS THE LAGLUTATIONOPEROSSIDASI DNA OXIDATION OF FREE RADICALS.

THE FUNCTIONS OF DNA

THE DNA IS THE MOLECULE THAT CONTAINS IF THE HERITAGE OF A PERSON: THE Genotype THAT A PERSON DIFFERENT FROM EACH OTHER AND MAKE A UNIQUE 'OF EACH INDIVIDUAL. THIS PROCESS IS OBTAINED THROUGH THE PROTEIN SYNTHESIS. NUCLEOTIDE SEQUENCE OF THE DIFFERENT AMINO must happen STYLING WHAT THE CONSTITUTION OF THE STRUCTURAL PROTEIN OF EACH PERSON. THE SYNTHESIS OF PROTEIN IS IN RIBOSOMES, organelles SURROUNDED BY CYTOPLASM DISCOVERED IN 1953 BY THE NOBEL PRIZE GEORGE PALADE.
The RIBOSOMES READING THE INFORMATION CONTAINED IN A CHAIN ??AMINO ACIDS DNA MESSENGER AND ASSOCIATE FOR PROTEIN SYNTHESIS IN A DIFFERENT MANNER TO A PERSON TO ANOTHER. DNA IN ITS ENTIRETY AND THE 'ESSENTIAL HEALTH PE, BECAUSE' THE COORDINATION STRUCTURE OF PROTEINS AND ENZYMES FROM WHICH DEPEND ON ALL FUNCTIONS 'LIVING ORGANISM.
FOOD ANTIOXIDANTS, AND THE AGE The pro-oxidant
PRODUCTS pro-oxidant (FOOD, DRUGS, CHEMOTHERAPEUTIC,, VITAMINS TYPE The ASCORBATE, METALS, The HYPERHOMOCYSTEINEMIA, ETC) induce OXIDATIVE STRESS IN TWO WAYS: 1) UP FREE RADICALS 2) REDUCE THE ANTIOXIDANTS. SEC. A RECENT STUDY UN. CLEVELAND USA (2013) FOODS RICH IN MOST ANTI-INFLAMMATORY AND ANTIOXIDANTS ARE: ALL THE PLANTS, FRUITS, HERBS, SPICES, WHEAT WHOLE, OLIVE OIL, NUTS, PULSES, SEEDS. EVEN THE OMEGA 3 FISH (HERRING, SALMON , mackerel, sardines, TUNA ETC.) exert anti-inflammatory action. A WIDE STUDY HAS SHOWN THAT WHO AVOID MEAT AND FOOD MADE FROM WHITE FLOUR HAS THE LOWEST LEVELS OF INFLAMMATION AND OXIDATION.
A DIET RICH IN FIBER REDUCES C REACTIVE PROTEIN OF 40% IN ACCORDANCE WITH THE SCHOLARS OF THE HARVARD REPORT GRAMS CARBOHYDRATES / FIBRE SHOULD BE IN GRAMS OF 8/1, STILL BETTER THAN 5/1. FOODS THAT PROMOTE INFLAMMATION ARE THE MEAT, THE SATURATED FAT, FAT OMEGA 6, THE REFINED CARBS, SUGAR, TRANS FAT IN FOODS THAT ARE DEVELOPED AND ALL OF FIBER FOODS impoverished.
MORE 'AND' HIGH CONSUMPTION OF THESE FOODS MORE 'AND' THE HIGH RATE OF C REACTIVE PROTEIN AND CYTOKINES.
A GREAT IMPORTANCE IN THE RELATIONSHIP BETWEEN FATTY OMEGA6/OMEGA3 feeding. THIS REPORT IS 'EQUAL TO 1/1 IN ALL MAMMALS AND IT WAS EQUAL TO 1 / I MAN PALEOLITHIC. Feeding MAN'S CURRENT REPORT AND '15/25 1.
A NEW STUDY OF 'AMERICAN HEART ASSOCIATION OF AUGUST 2013 HAS CONCLUDED THAT THE IMPACT DICTUS IN WOMEN AND' PROPORTIONAL TO REVERSE UNIT 'ORAC ASSUMED. 36000 WOMEN WERE FOLLOWED FOR MANY YEARS.(Rautianen S.ET ALL.STROKE 43 (2) :332-340/2012).
THE PREVALENCE OF FATTY FOOD POLIINSATURIOMEGA 6 E'INDICATA TO CAUSE CANCER, ARTHRITIS, HEART ATTACK, STROKE HYPERTENSION ETC.
SECOND WORK OF THE CENTER FOR Sinopoulos GENETIS U.S. A REPORT 4/1 OMEGA MORTALITA'CARDIOVASCOLARE REDUCE BY 70%.
A REPORT OMEGA 6/3 TO 2/1 REDUCES THE RISK OF CANCER OF THE COLON AND ABOLISH INFLAMMATION IN PATIENTS WITH ARTHRITIS REUMATOIDE.UN RATIO 5/1 IMPROVE ASTHMA IN WOMEN BETTER WHEN THE RISK OF BREAST CANCER. (BIOMEDPHARMACOTHER, 2002 OCT; 56/8 :365-7 THE AGE (ADVANCED glycosylation END PRODUCTS) AND PROTEIN CROSS-LINKING.
MAN THE AGE OF COOKED FOOD HIGH TEMPERATURE ARE GREAT FONTEDI FREE RADICALS. THE AGE (ADVANCED GLYCATIONENDPRODUCTS) ARE DUE TO glycation, REACTION IN WHICH THE MOLECULES OF SUGAR REACT IN OR OUT OF THE BODY WITH PROTEIN, originating OF denatured proteins, CROSS LINKING PROTEIN.
The END PRODUCTS ARE FREE RADICALS GENERATED BY AGE OR OR beta amyloid proteins denatured proteins, IRREVERSIBLE.
Glycation AND 'NON-REACTION ENZIMALICA, INSIDE THE BODY DEPENDS ON THE HIGH GLUCOSE, FUORIDAL'ORGANISMO ARE NEEDED FOR HIGH TEMPERATURES AND PROLONGED glycation.
THE TEMPERATURE OF BAKING AND COOKING TIME AFFECT THE AMOUNT 'OF AGE.
THE FORMATION OF AGE IS IN 3 STEPS:

  • FORMATION OF A SCHIFF BASE, AND THAT 'DUE TO A DOUBLE BOND BETWEEN THE CARBON ATOM OF GLUCOSE AND' NITROGEN ATOMS OF LYSINE.
  • AMADORI FORMATION OF PRODUCTS IN THIS CASE AN ATOM OF HYDROGEN CONNECTS TO GRUPPONITROGENO, CREATING A KETONE.
  • LA TRAINING rad IAGEs, THROUGH THE OXIDATION OF AMADORI PRODUCTS, WHICH ARE IRREVERSIBLE.

DIETS OF CURRENT SATURATED COOKED FOOD FOR HIGH TEMPERATURE CONTAIN LARGE QUANTITY OF AGE. AGEs CAUSING THE CURRENT OUTBREAK OF CANCER, DIABETES AND HEART DISEASE. COOKED FOODS CONTAIN HIGH TEMPERATURE UP TO 100 TIMES MORE AGE OF RAW FOODS. FOODS RICH IN ANIMAL FATS AND PROTEINS AFTER MANY MORE COTTURASVILUPPANO 'AGE OF VEGETABLE FOODS (FRUITS, VEGETABLES, MILK AND YOGURT).
THE COMPLICATIONS OF DIABETES (REINOPATIE, coronary artery disease, INSUFF.RENALI, ETC ARE TO BLAME AGEs. Glycation THE REACTION OF DISCOVERY WAS IN 1912 BY MAILLARD, BUT 'was underestimated IN TIME. Maillard reaction E' DUE TO THE REACTION OF AMINO ACID AND GLUCOSE AND OUT CALLS IN BY HIGH TEMPERATURE.
CREATE THIS REACTION OF AGE AND 'HEAD OF COLORS AND SCENTS OF MANY PRODUCTS: MEAT ON THE GRILL, TOAST, BISCUITS, FRENCH FRIES, BEER AND WHISKEY, FRIED ONIONS, CONDENSED MILK, COFFEE', THE CRUST OF CAKE, BISCUITS, CAKES , BREAD AND MAPLE SYRUP. AGE IS THE LINK TO THE CELL MEMBRANES IN SPECIFIC RECEPTORS (OR RAGE receptor for age) CAUSING THE FORMATION OF FREE RADICALS OF OXIDATIVE STRESS AND INFLAMMATION. THE AGE MAY OCCUR EVEN AFTER THE FATS ARE CALLED ALES AND HAVE A BIG SOURCE OF FREE RADICALS.

GLIAGEs CAUSES MANY DISEASES:

ALZHEIMER'S DISEASE, CANCER, HEART DISEASE, DIABETES 2, kidney disease, atherosclerosis, hypertension, stroke, SKIN DISEASES, DISORDERS VISUAL. THE ABILITY TO 'AND DESTRUCTIVE OF AGES' HUGE FACT GLIAGEs DAMAGED PROTEINS, DNA and RNA. GLIAGEs ENDEAVOUR SUCH DAMAGES WITH THE SYNTHESIS OF PROTEIN OXIDIZED, MAL WORKING AS DEFINED CROSS-LINKING. PROTEIN CROSS-LINKING ENDEAVOUR CATARACT, ALZHEIMER'S, PARKINSON'S AND ALS.1. THE SOURCE OF AGEsE 'INTERNAL AND EXTERNAL IN BOTH CASES AND' DUE TO THE REACTION BETWEEN CARBOHYDRATE AND PROTEIN.
Glycation CAN 'TAKE PLACE IN THE BODY OR OUT IN BY ENDOGENOUS Glycation Glycation exogenous glycated ARE EXAMPLES OF FOOD: BISCUITS, BREAD. MEAT ON THE GRILL, FRIES ETC. Glycation ENDOGENOUS IS IN THE BODY FROM GLUCOSE, FRUCTOSE FROM FROM GALACTOSE. FRUCTOSE AND GALLATTOSIO glycan 10 TIMES MORE 'OF GLUCOSE. Glycation ENDOGENOUS AND 'frequent IN DIABETIC AND CAUSE THE COMPLICATIONS OF DIABETES. The AGEs MOST COMMON AND 'THE GLUCOSOEPANE (WITH FORMULA C18H32N6O6) A PROTEIN THAT IS FORMED BY THE REACTION OF GLUCOSE WITH AMINO ACIDS LYSINE AND ARGININE.
IN THE HUMAN TISSUE AND GLUCOSOEPANE '10 TO 1000 TIMES MORE' TOWN OF OTHER AGE. IN COLLAGEN AND THE LENS WITH ADVANCING AGE 'YOU HAVE A SIGNIFICANT AMOUNT OF ACCUMULATION OF GLUCOSOEPANE'.
COOKING LIVE FIRE (225 °C), frying (177°C) COOKING ROAST (177 °C), THE BOILING (100°C) DETEMINANO THE GREATER QUANTITY 'DIAGEs. REDUCE SUGAR FOOD COOK FOOD FOR A SHORT TIME AND TEMPERATURE LOW, DRINK A LOT OF WATER AND TAKE VITAMIN SUPPLEMENTS B6 (PIRIDOXAMINA) PROTECT THE FORMATION OF AGE.
REDUCED THE INTRODUCTION OF FOOD AGEs PREVENTS HEART DISEASE, THE KIDNEY, DIABETES 1e2, IMPROVED SENSITIVITY 'insulin AND LEADS THE HEALING OF WOUNDS.
LOW THE INTRODUCTION OF AGE
EXTENDS LIFE AS THE CALORIE RESTRICTION IN MICE. (SOURCE American Dietetic Association 2010).
CONTENTS BY AGE FOR SOME FOOD FOR 100 GR.
FRUIT
APPLE = 45
BANANA = 9
DATES = 60
PRUNES = 167
VEGETABLES
CARROTS = 10
CELERY = 43
GREEN BEANS IN BOX = 18
ONIONS = 36
Pomidori = 23
CUCUMBER = 31
DAIRY.
SKIMMED = 1
ICE = 34
CHERRY YOGURT = 4
VANILLA YOGURT = 3
HONEY = 7
MEAT
HAM FRIED 5 MINUTES = 91577
ROAST PORK = 3544
LAMB GIGOT BOILED 30 MINUTES = 1218
ROAST CHICKEN = 8802
BATTERED CHICKEN BREAST = 4938
CHICKEN BREAST IN THE OVEN 25 MINUTES WITH SKIN = 9961
VEAL STEAK PANATA = 10058
VEAL MEATBALLS = 4300
FATS
BUTTER = 26480
MARGARINE = 17520
MAYONNAISE = 9400
PEANUTS ARR. = 6447
OLIVE OIL = 11900
SESAME OIL = 21680
FISH
SALMON = 1212
TROUT BAKED = 2138
BAKED TUNA = 919
GRILLED TUNA = 5150
CANNED TUNA, WATER = 452
CHEESE =
FETA = 8423
GRATED PARMESAN = 16900
CHEDDAR = 5523
COTTAGE = 1453
CRACKER
POTATO CHIPS = 2883
FOR MORE ':
SEARCH IN THE WORLD PORTALS: 1) AGE IN FOODS. 2) GLUCOSOEPANE, 3) MAILLARD REACTION PRODUCTS.

CARNITINE AND DNA

CARNITINE AND THE 'A SMALL MOLECULE SOLUBLE NORMALLY PRODUCED IN BY A PARTY REDAGLI Amino acids methionine and lysine.
THE DAILY PRODUCTION AND 'OF 80 MG PROKG PRO DIE. THE SYNTHESIS OF CARNITINE AND DECREASES 45/50ANNI CROLLA AFTER 70 YEARS. CARNITINE AND THE 'ESSENTIAL FOR ENERGY PRODUCTION IN THAT EASY ENTRY OF LONG CHAIN ??FATTY ACID EXCRETION The NEIMITOCONDRI EASY DEL'ACETILCOENZIMA AE IMPROVES FINALLY THE SYNTHESIS OF atypical.
The Acetylcarnitine LIPOIC ACID ASSOCIATED SPECTACULAR RESULTS OBTAINED IN THE IMPROVEMENT OF OLD RATS, AT BRAIN, AND CARDIAC MUSCLE. (PROF B. AMES, Univ. OF BERKELEY). IN A JOB AND WITH Abdelrazik. UN.CLEVELAND 420 OF 2009 ON EMBRYOS OF THE EFFECTIVENESS OF RATS has been demonstrated Acetylcarnitine THE PROTECTION OF DNA BY ROS.
Acetyl Carnitine THEREFORE THE EXCELLENT RESULTS IN DNA REPAIR IN produzionedi ENERGY IN THE REPAIR OF OXIDATIVE DAMAGE ASSOCIATED SPECIES ALL'ACIDOLIPOICO.

OXIDATION PRODUCTS OF DNA

THE MAJORITY OF OXIDATION PRODUCT OF DNA AND 'THE 8-O OXOdG DEOXIGUANOSINA the concentrations of 8-OXO-DG INSIDE MOBILE MEASURE OXIDATIVE STRESS. DEOXIGUANOSINA IS INCREASED IN THE CELLS OF PERSONS SUFFERING FROM CANCER. GLUTATHIONE AND ASCORBATE (vit.C) ARE VERY EFFECTIVE IN DNA REPAIR AND OXIDATION AND REDUCTION DEOXIGUANOSINA IN THE DNA OF HUMAN LYMPHOCYTES. (P DM 10,223,188) NUMBER OF AUTHORS ACCORDING TO THE OXIDATION OF DNA METABOLISM OR PRODUCED BY RADIATION OR CHEMICALS,
HELPS IN THE DECISIVE onset CANCER. THE HUMAN DNA IS NOT 'an inert substance, REACTIVE ENVIRONMENT OF THE CELL, THE PRESENCE OF TOXIC SUBSTANCES AND THE PRESENCE OF ULTRAVIOLET RADIATION RADIATION AND MAKE THEM LIABLE TO INSULT that oxidize BASES AND ALTER THE SUGAR-PHOSPHATE GROUPS.
THE DNA REPAIR AND 'AN ESSENTIAL PROCESS FOR THE SURVIVAL WHY' MUST PROTECT THE GENOME CHANGES FROM AND MUST MAINTAIN UNIFORM WITH PRECISION 'PROTEIN IN INDIVIDUALS. DNA DIRECTED IN FACT THE LIVING THE SYNTHESIS OF PROTEIN AND AN ERROR HAS SERIOUS CONSEQUENCES.
THE DIRECTIONS GIVEN BY THE DNA polypeptide chains REQUIRES THE PRESENCE OF TWO PROJECTS: TRANSCRIPTION AND TRANSLATION AND THE PRESENCE OF A MESSENGER RNA ribonucleic acid O.
NUCLEOTIDE SEQUENCE OF THE DIFFERENT BETWEEN THE INDIVIDUALS MUST INDICATE WHICH AMINO ASSOCIATE IN THE SYNTHESIS OF PROTEIN. THE SYNTHESIS OF PROTEIN IS IN RIBOSOMES, organelles IMMERSINE CYTOPLASM: The RIBOSOMES READING THE
INFORMATION IN A CHAIN ??OF MESSENGER RNA (m-RNA). THE TRANSCRIPT IS IN THE READING OF THE DOUBLE HELIX OF DNA SEQUENCES IN THE SEQUENCE OF A SINGLE PROPELLER RNA. THE TRANSLATION IS IN CYTOPLASM WHERE YOU ASSOCIATE TO THE MRNA RIBOSOMES, DE CODONS ALLOW THE READING OF MRNA.

GENOME

THE GENOME OF A BODY AND 'MADE UP OF PAIRS OF SEQUENCES BASED DISTRIBUTED IN CHROMOSOMES, ONLY SOME COUPLES ARE IN TRANSCRIBED RNA
. GENES ARE COUPLE OF BASES THAT ARE IN TRANSCRIBED RNA, THIS PROCESS IS CALLED GENE EXPRESSION.

DNA

CELLS HAVE TWO DNA

1) THE MITOCHONDRIAL
2) THE NUCLEAR.

THE NUCLEAR AND 'THE CORE CONTENT, SURROUNDED BY A MEMBRANE THAT THE CAPSULES DNA (deoxyribonucleic acid).
THE NUCLEAR DNA AND 'THE CUSTODIAN GENETIC INFORMATION CONTAINED IN SEQUENCE OF BASES OF DNA MOLECULES, LOCATED IN CHROMOSOMES. GENES ARE MADE OF PORTIONS OF DNA AND THE GENOME AND 'THE SUM TOTAL OF THE DNA OF THE ORGANISM.
THE HUMAN GENOME AND 'KEPT IN 23 PAIADICROM OSOMI CONTAINS APPROXIMATELY 23000 PROTEIN GENES ENCODING.
THE NUMBER OF GENES, CONTAINED IN THE HUMAN GENOME AND 'WAS REBUILT piu'volte, TODAY IS ESTIMATED THAT THE HUMAN CELLS CONTAINING APPROXIMATELY 20000/25000 GENES. The AGING FOR THE DNA DAMAGE CAUSED BY FREE RADICALS AND OXIDATIVE DAMAGE IF YOU SEND WRONG SIGNALS FOR THE PRODUCTION OF ENZYMES, THE COENZYMES, PROTEIN, FAT AND HORMONES.
FREE RADICALS ALSO CAUSE MUTATIONS.
THE CHANGES OF HUMAN DNA IS DUE TO EXCHANGE OF MOLECULES THAT ARE: The NUCLEOTIDES.
THE CHANGES OF DNA REPAIR IS NOT DURING THE CELL REPLICATION TRANSMITTED TO THE DAUGHTERS OF BECOMING POSSIBLE CAUSE CARCINOMAS. THE DNA DAMAGE TO RESULT IN A STEADY DECLINE OF HEALTH AND VITALITY 'IN THE ELDERLY AND THE OXIDATIVE DNA DAMAGE MUTATION ACTS, DYSFUNCTIONS senile, INCREASED RISK OF CANCER.
THE PERCENTAGE OF OXIDATION OF CELLULAR STRUCTURES AND 'WAS STUDIED IN RATS OF DIFFERENT AGE': IN RATS OF 29 MONTHS oxidized proteins UP BY 150% IN RELATION TO PROTEIN OF RATS OF 12 MONTHS, THE FATS OXIDISED UP BY 267% IN RATS OF 24 MONTHS WITH RESPECT TO THE RATS OF 7 MONTHS.